![]() Wellbeing Hub ramps up health services – launches blood pressure checks Goes to them, why and what benefits they get from participating. “define new frontiers in the science and health optimisation for the next generation.”ĭestination spas fund study examining experiences of 2,600 health retreat guestsĭespite the growing popularity of health retreats globally, little to no research explores who The research has not yet been peer-reviewed, but has been published: LATEST NEWSĮquinox announces new Health Advisory Board to elevate its programmesĮquinox, has appointed a Health Advisory Board of top medical experts and academics to The research has not yet been peer-reviewed, but has been published: Researchers at Stanford found fat cells can be infected by COVID-19, provoking to a ‘robust inflammatory response’ Credit: photo: Shutterstock/UGREEN 3S We further demonstrate that SARS-CoV-2 RNA is detectable in adipocytes in COVID-19 autopsy cases and is associated with an inflammatory infiltrate. “Preadipocytes, while not infected, adopt a pro-inflammatory phenotype. “Adipose tissue macrophage infection is largely restricted to a highly inflammatory subpopulation of macrophages, present at baseline, that is further activated in response to SARS-CoV-2 infection. “We identify two cellular targets of SARS-CoV-2 infection in adipose tissue: mature adipocytes and adipose tissue macrophages. “We demonstrate that human fat tissue is ‘permissive’ to SARS-CoV-2 infection – the virus that causes COVID-19 – and that infection elicits an inflammatory response, including the secretion of known inflammatory mediators of severe COVID-19. ![]() ![]() In reporting the details of the findings, the researchers said: “Collectively, our findings indicate that adipose (fat) tissue supports SARS-CoV-2 infection and pathogenic inflammation and may explain the link between obesity and severe COVID-19.“Obesity is associated with adverse COVID-19 outcomes, but the underlying mechanism was unknown up to this point. In addition to explaining why patients with excess body weight are particularly vulnerable, it may also explain why some younger adults with no underlying health issues become so ill. The findings show the virus is able to evade the immune defences within the body’s fat cells, before causing inflammation elsewhere in the body. It found that fat cells and also immune cells (macrophages) can be infected, leading to a ‘robust inflammatory response’. The study, led by Stanford University School of Medicine, examined whether fat tissue obtained from bariatric surgery patients could become infected with the SARS-CoV-2, and tracked how various types of cells responded to the virus. ![]() The study lends added credibility to the work being done by spa and wellness businesses in supporting people to control their levels of body fat. New research suggests that COVID-19 infects fat cells, explaining why overweight and obese are at a higher risk of severe illness and death from COVID-19. Researchers at Stanford found fat cells can be infected by COVID-19, provoking to a ‘robust inflammatory response’ Credit: photo: Shutterstock/UGREEN 3S ![]()
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